rockefeller university press

(Rockefeller University Press) The overlooked and undervalued protein, sarcospan, just got its moment in the spotlight.

Peter et al., reporting in the Journal of Cell Biology, now show that adding it to muscle cells might ameliorate the most severe form of muscular dystrophy.

(Rockefeller University Press) Amyloid precursor protein, whose cleavage product, amyloid-b, builds up into fibrous plaques in the brains of Alzheimer's disease patients, jumps from one specialized membrane microdomain to another to be cleaved, report Sakurai et al in the Journal of Cell Biology.

(Rockefeller University Press) Fritz et al., reporting in the Journal of Cell Biology, have identified an amino acid switch that flaviviruses flip to gain access to cells.

(Rockefeller University Press) By mopping up excess neurotrophic factor from neuronal synapses, astrocytes may finely tune synaptic transmission to affect processes such as learning and memory, say Bergami et al.

Their report is published in the Journal of Cell Biology.

(Rockefeller University Press) New research may help reveal why different parts of the brain can come under attack in patients with multiple sclerosis.

According to a new study in mice with an MS-like disease, the brain's response to a protein produced by invading T cells dictates whether it's the spinal cord or cerebellum that comes under fire.

The study will be published online on Oct. 13 in the Journal of Experimental Medicine.

(Rockefeller University Press) A potentially blinding neurological disorder, often confused with multiple sclerosis, has now become a little less mysterious.

A new study by researchers at the Mayo Clinic in Rochester, Minnesota, may have uncovered the cause of Devic's disease.

Their new study, which will appear online on October 6th in the Journal of Experimental Medicine, could result in new treatment options for this devastating disease.

(Rockefeller University Press) A cell decides what proteins to make based on the messages it receives from its genome.

Sometimes messages are held back to be read later, and in most cell types these delayed messages are stored and marked for destruction in P bodies.

P bodies in worm egg cells, however, are message protectors, according to a paper by Boag et al. appearing in the Aug. 11 issue of the Journal of Cell Biology.